ECG Interpretation: cram pack

Formula sheets, the key method steps for every topic and 4 practice problems, with full solutions at the back.

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1Formulas and methods

ECG rhythm strip interpretation

Paper (25 mm/s, 10 mm/mV)
1 small box = 0.04 s, 1 large box = 0.20 s, 10 mm = 1 mV
Heart rate, regular rhythm
300 ÷ large boxes = 1500 ÷ small boxes = 60 ÷ RR (s)
Heart rate, irregular rhythm
QRS complexes in 6 s × 10
Normal PR interval
0.12–0.20 s
Normal QRS duration
< 0.12 s
QTc (Bazett)
QT/√RR
QTc (Fridericia)
QT/∛RR
QTc (Framingham)
QT + 154·(1 − RR)
Sinus rates
bradycardia < 60, normal 60–100, tachycardia > 100 /min
Escape rates
junctional 40–60, ventricular 20–40 /min
Fast rhythms
SVT 150–250 /min (narrow), VT > 100 /min (wide)

Method

  1. Rate: for a regular rhythm divide 300 by the large boxes or 1500 by the small boxes between two R waves; for an irregular rhythm count the QRS complexes in 6 seconds and multiply by 10.
  2. Rhythm: compare the R–R intervals. Equal is regular; a repeating pattern is regularly irregular; no pattern at all is irregularly irregular.
  3. P waves: is there an upright P wave in lead II before every QRS, all alike? Look for fibrillatory or sawtooth waves, inverted P waves and P waves without a QRS.
  4. PR interval: from the start of P to the start of the QRS, normally 0.12–0.20 s and constant. Watch whether it lengthens, stays fixed before a dropped beat, or varies at random.
  5. QRS duration: below 0.12 s is narrow; 0.12 s or more is wide. Then name the rhythm, and for the QT use QTc = QT/√RR (Bazett) with times in seconds.

2Practice problems

1.ECG rhythm strip interpretationMedium
II25 mm/s · 10 mm/mVnormal sinus rhythmsinus tachycardiajunctional rhythmsinus bradycardia

Interpret this lead II rhythm strip. Which rhythm is it? Click your answer.

lead II, 25 mm/s, 10 mm/mV1 small box = 0.04 s, 1 large box = 0.20 sstrip length 6 s
  1. 1normal sinus rhythm
  2. 2sinus tachycardia
  3. 3junctional rhythm
  4. 4sinus bradycardia
2.ECG rhythm strip interpretationHard
II25 mm/s · 10 mm/mVsecond-degree AV block type IIidioventricular rhythmdual-chamber (AV) paced rhythmpaced rhythm, failure to capture

This patient has a pacemaker. Interpret the lead II strip. Click your answer.

lead II, 25 mm/s, 10 mm/mV1 small box = 0.04 s, 1 large box = 0.20 sstrip length 6 s
  1. 1second-degree AV block type II
  2. 2idioventricular rhythm
  3. 3dual-chamber (AV) paced rhythm
  4. 4paced rhythm, failure to capture
3.Human physiologyMedium

Where is the heart's natural pacemaker?

  1. APurkinje fibres
  2. BBundle of His
  3. CAtrioventricular node
  4. DSinoatrial node
4.Human physiologyHard

Why is severe hyperkalaemia dangerous for the heart?

  1. AIt speeds up the SA node until tachycardia causes fainting
  2. BIt hyperpolarises cardiac cells, so they can never fire
  3. CIt blocks the binding of calcium to troponin
  4. DIt depolarises cardiac cells, inactivating sodium channels and causing arrhythmias

3Solutions

1.ECG rhythm strip interpretation

Answer: sinus bradycardia

Rhythm criteria (lead II): sinus rhythm has an upright P wave before every QRS, a constant PR of 0.12–0.20 s and a narrow QRS: normal sinus rhythm at 60–100/min, sinus bradycardia below 60, sinus tachycardia above 100. Atrial fibrillation: no P waves, a fibrillatory baseline and an irregularly irregular R–R. Atrial flutter: sawtooth flutter waves at about 250–350/min with a fixed conduction ratio such as 2:1 or 4:1. First-degree AV block: sinus rhythm with a constant PR above 0.20 s. Junctional rhythm: narrow QRS at 40–60/min with absent or inverted P waves (PR below 0.12 s, or P after the QRS). A PVC is an early, wide (0.12 s or more), bizarre QRS without a P wave, with the T wave in the opposite direction and usually a full compensatory pause; in bigeminy every second beat is a PVC.

Five steps

1. Rate: R–R = 31 small boxes, 1500 ÷ 31 ≈ 48 per minute.
2. Rhythm: regular, every R–R is 1.24 s.
3. P waves: upright, one before every QRS, all alike.
4. PR interval: 0.14 s, constant, within 0.12–0.20 s.
5. QRS: 0.08 s, narrow (below 0.12 s).

Interpretation

sinus bradycardia

Answer

sinus bradycardia
2.ECG rhythm strip interpretation

Answer: paced rhythm, failure to capture

Paced rhythms: a pacemaker impulse shows as a narrow vertical pacing spike. After a ventricular spike the QRS is wide (0.12 s or more), because the impulse spreads from the lead through the muscle instead of the conduction system; with right-ventricular pacing it is often mostly negative in lead II. A spike before each P wave means atrial pacing, and a dual-chamber (AV) pacemaker shows an atrial spike and a P wave, then, after the programmed AV delay, a ventricular spike and a wide QRS. Capture means every spike is followed by a depolarisation. Failure to capture is a spike without a P wave or QRS after it; failure to sense (spikes that ignore the patient’s own beats) and failure to pace (no spike when one is due) are the other classic malfunctions.

Five steps

1. Rate: 5 QRS complexes in 6 s, × 10 = 50 per minute.
2. Rhythm: the pacing spikes are regular (every 0.92 s), but the R–R is not, because a QRS is missing after some spikes.
3. P waves: none visible.
4. PR interval: not measurable.
5. QRS: 0.14 s and wide after the captured spikes; one spike is not followed by a QRS (no capture).

Interpretation

paced rhythm, failure to capture

Answer

paced rhythm, failure to capture

Failure to capture: the pacemaker fires but the heart does not respond. Textbooks treat it as urgent to report, because the patient may depend on the pacemaker.

3.Human physiology

Answer: D · Sinoatrial node

Why this is the answer

The SA node depolarises spontaneously at the fastest rate and sets the heart rhythm. The AV node delays the impulse before it spreads to the ventricles.

Answer

D · Sinoatrial node
4.Human physiology

Answer: D · It depolarises cardiac cells, inactivating sodium channels and causing arrhythmias

Why this is the answer

High extracellular potassium makes the resting potential less negative. Many fast sodium channels then become inactivated, slowing conduction and risking fatal arrhythmias.

Answer

D · It depolarises cardiac cells, inactivating sodium channels and causing arrhythmias

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